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Vitamin A supplementation in patients with
low vitamin A levels resulted in increased interleukin-10
(IL-10) and decreased tumour necrosis factor-alpha
(TNFalpha) levels, as found in both plasma and monocyte
supernatants, possibly favouring anti-inflammatory net effects.
Decreased vitamin A levels in common variable
immunodeficiency: vitamin A supplementation in vivo enhances
immunoglobulin production and downregulates inflammatory responses.